Roger Davis’ studies of signal transduction mechanisms led to the molecular cloning of human cJun NH2-terminal kinase (JNK), a founding member of the stress-activated MAP kinase group.  The subsequent focus of his research program has been to understand the molecular basis for the function of JNK in both physiology and disease processes.  The overall goal of Dr Davis' research is to design novel therapeutic strategies for the treatment of inflammation-related disease.  

Dr Davis was elected to the National Academy of Sciences, National Academy of Medicine, National Academy of Inventors, American Academy of Arts & Sciences, American Academy of Microbiology, American Association for the Advancement of Science, European Molecular Biology Organization, and The Royal Society.  

Davis Lab